VIM and lysosomal lipid storage disorder: In our study, it was demonstrated that stable atherosclerotic plaques at the stage of lipidosis and fibrosis were characterized by increased amounts of cytoskeletal proteins such as actin, tropomyosin, tubulin, MAGP-4, and vimentin; this alteration is apparently associated with cellular migration to the subendothelial space after damage to the vessel wall (during subsequent inflammation).