More importantly, we observed that MYC protein was reduced in LINC01116‐silenced NPC cells due to hindered interaction between LINC01116 and MYC mRNA (Figure S2A,B); conversely, such interactivity was encouraged and therefore MYC protein expression elevated in HONE1 and CNE1 cells under LINC01116 overexpression (Figure S2C,D), suggesting LINC01116 affected MYC protein through its interaction with MYC mRNA. Here, MYC is linked to nasopharyngeal carcinoma.