The PAMS in Crb-deficient SG cells have striking similarities to the inclusion bodies observed in MVID patients carrying mutations in MYO5b (Müller et al., 2008; Ruemmele et al., 2010) or those found in animal models of MVID, like zebrafish mutant for myosin Vb (Sidhaye et al., 2016) and mice mutant for Rab8a and Rab11a (Feng et al., 2017; Sato et al., 2007). Here, RAB11A is linked to microvillus inclusion disease.