FUS and infection: We observed that iPSC-derived SNs with WT FUS-eGFP showed increased total FUS-eGFP levels (cytoplasmic + nuclear FUS-eGFP) following RABVΔG infection compared with uninfected controls (RABVΔG total: LL WT uninfected vs. LL WT infected, p < 0.01, 1way ANOVA, Tukey post-test for multiple comparisons; Figure 5D), suggesting that RABVΔG infection caused defects in FUS turnover.