Further evidence that pMo contribute to the kidney pathology is suggested by the recent finding that chimaeric mice devoid of pMo, which were generated by injecting liver cells from the foetus of ABIN1 KO × Nr4a1 KO mice into irradiated WT mice, did not develop glomerulonephritis (Kuriakose et al, 2019). The gene discussed is NR4A1; the disease is glomerulonephritis.