The loss of CCL5 ultimately led to a switch in bacteria from carrier state to lethal state of infection.30 CCL22 is known to promote Th2‐mediated immune responses such as airway hypersensitivity, atopic dermatitis, and eosinophilic pneumonia,31, 32, 33 but its role in bacterial infections is unclear. The gene discussed is CCL22; the disease is atopic eczema.