Besides, the phosphorylation of Tyr1068 in EGFR, Ser473 in AKT and Thr202/Tyr204 in Erk1/2 were declined in three GBM cell lines after MYST1 silencing, compared with GFP silencing, indicating EGFR‐AKT‐ERK1/2 signaling pathway was downregulated by MYST1 knockdown (Figure 5F). The gene discussed is EGFR; the disease is glioblastoma.