EZH2 and gastric cancer: It promoted cell invasion and migration by increasing MMP2 and MMP9.78SNHG17 bound with EZH2 and inhibited the expression of CDKN2B and CDKN1C to promote gastric cancer cell cycle progression in the G0/G1 phase.26 Liu et al further proposed that SNHG20 could facilitate gastric cancer cell proliferation and invasion.35 Another study showed SNHG20 was located in the cytoplasm, and SNHG20 interacted with MIR‐495‐3p to upregulate ZFX, and promoted gastric tumor growth and invasion.79