Li et al proposed that SNHG5 could upregulate CTNNB1, MYC and CCND1 expression to activate the Wnt signaling pathway and then induce Epithelial‐mesenchymal transition (EMT) to promote liver cancer cell invasion.25SNHG17 bound with EZH2 and inhibited the expression of CDKN2B and CDKN1C to promote gastric cancer cell cycle progression.26 However, since the biofunctions, molecular mechanisms and potential pathways of SNHGs in digestive cancers are complicated, and they have not yet been clearly defined. This evidence concerns the gene EZH2 and gastric cancer.