SNHG3 acted as an oncogene and accelerated cancer cell growth in tumorigenesis.47 In colorectal cancer, SNHG3 could augment the expression of MYC and MYC’s target genes CCNB1, CCND2, CDK4 and E2F1. Combining the results of GSE54632 database and starBase2.1, Huang et al found that MIR‐182‐5p was the only gene that could target MYC and at the same time, be sponged by SNHG3. SNHG3 obstructed MIR‐182‐5p's suppression on MYC to promote tumor growth.47 This evidence concerns the gene MYC and cancer.