GSK3B and neoplasm: The overexpression of SNHG3 predicted high rates of larger tumor size, portal vein tumor thrombus, sorafenib resistance and relapse.91, 92 It directly combined with MIR128 to upregulate the expression CD151, and activated EMT to promote cell invasion.91 Li found that the overexpression of SNHG5 inhibited the suppressive influence of MIR‐26a‐5p on GSK3B to promote liver cancer tumorigenesis.