CYP2E1 and metabolic dysfunction-associated steatohepatitis: In NASH-induced dysbiosis, conversely, the over-representation of alcohol-producing bacteria determined an exaggerated release of ethanol into the blood flow, further corroborating liver inflammation, reactive oxygen species (ROS) production via the Cytochrome P450 2E1 (CYP2E1) [57,60] and intestinal hyperpermeability [57].