CrPV, a member of the Dicistroviridae family, selectively disrupts GW182/Dcp1 but not Ago1/Ago2 aggregates, suggesting that they play differential roles during infection (Khong and Jan, 2011), while that RNAs of SINV, a virus that belong to the Togaviridae family, interact with HuR in order to stabilize and avoid the cellular mRNA decay machinery (Sokoloski et al., 2010). This evidence concerns the gene DCP1B and infection.