The objective of this study was to investigate, using an allergic murine model of asthma, whether the Ang-1(1–7)/MAS1 receptor axis a) inhibits allergic inflammation via modulation of Src kinase and/or EGFR and/or their downstream signaling pathways through ERK1/2 and b), modulates neutrophil and/or eosinophil chemotaxis ex vivo to bronchoalveolar lavage (BALF) from OVA challenged mice. This evidence concerns the gene EGFR and asthma.