However, it was notable that the electroencephalogram in patient 2 showed persistent burst‐suppression patterns even beyond 3 years of age, which has not so far been described in cases of early infantile epileptic encephalopathy caused by KCNQ2 or SCN2A mutations.23 Further investigations in a larger cohort of patients are warranted to explore characteristic electroencephalographic progression and optimal antiepileptic strategies for patients with NSF‐related early infantile epileptic encephalopathy. This evidence concerns the gene SCN2A and genetic developmental and epileptic encephalopathy.