Due to the fact that a specific early event during FCV infection such as binding/entry was altered in cells overexpressing survivin or treated with the conditioned medium from these cells; but the infection with MNV-1, another calicivirus that has a different surface receptor than FCV, was not altered [21,22], suggested that the overexpression of survivin could particularly affect the levels or the subcellular localization of fJAM-1, the FCV functional receptor. The gene discussed is BIRC5; the disease is infection.