Therefore, it is possible that the overexpression of survivin in RAW 267.4 and CrFK cells emulates this condition of the high abundance of intracellular survivin observed in cancer cells and, consequently, the delivery of exosomal survivin into the culture media, which may explain the reduction in the FCV infection of cells treated with the supernatants from cells overexpressing survivin. Here, BIRC5 is linked to cancer.