Through repressing c-Jun N-terminal kinase (JNK)/nuclear factor-κB (NF-κB) pathway and binding to tumor necrosis factor-α (TNF-α), lncRNA plasmacytoma variant translocation 1 (PVT1) may enhance inflammatory response in lipopolysaccharide (LPS)-induced septic AKI [13]. The gene discussed is NFKB1; the disease is acute kidney injury.