It is possible that the increased IFN-α/β production by WT may be due to changes in miR-142-3p levels within the PLN as a response to infection, leading to virus replication or, possibly, virus escape of miR-142-3p suppression in myeloid cells due to the presence of higher levels of WT virus in the PLN at 24 hpi compared to 12 hpi [30]. This evidence concerns the gene IFNA1 and infection.