Levels of CXCL8/IL‐8, IL‐13, CCL11/eotaxin, CXCL10/IP‐10, CCL3/MIP‐1α, CCL4/MIP‐1β, and TNF‐α showed positive correlations with CSF protein concentrations in NF155+ CIDP patients (CXCL8/IL‐8, r = 0.4860, P = 0.031; IL‐13, r = 0.5455, P = 0.0007; CCL11/eotaxin, r = 0.6347, P < 0.0001; CXCL10/IP‐10, r = 0.6003, P = 0.0001; CCL3/MIP‐1α, r = 0.4486, P = 0.0069; CCL4/MIP‐1β, r = 0.4132, P = 0.0136; TNF‐α, r = 0.3741, P = 0.0268) (Fig. 2B), while IL‐1ra showed a negative correlation with CSF protein amounts (r = −0.4278, P < 0.0104). This evidence concerns the gene CXCL8 and chronic inflammatory demyelinating polyradiculoneuropathy.