They demonstrated that the most dominant phenotype in the circulation (i.e., PBMCs) and tumor-infiltrating pancreatic tissues were G-MDSCs (Lin-HLA-DR-CD33+CD11b+CD15+) and not M-MDSCs (Lin-HLA-DR-CD14+) when compared with chronic pancreatitis patients and healthy donors [15]. The gene discussed is FUT4; the disease is neoplasm.