With the knowledge of species-specific DENV-mediated antagonism of the cGAS-STING pathway [74], Ding et al. infected a panel of dermal fibroblasts isolated from humans, non-human primates, and mice with ZIKV and found that human and non-human primate fibroblasts were similarly permissive to ZIKV infection, while mouse fibroblasts were comparatively resistant [61]. The gene discussed is CGAS; the disease is Zika virus infectious disease.