Both in breast cancer (BC) tissue samples and cell lines, TP73-AS1 has been found to be up-regulated and this overexpression has been related to several BC clinicopathologic characterizations, such as TNM stage, tumor size, lymph node metastasis, and oOS In contrast, TP73-AS1 silencing suppressed vasculogenic mimicry, invasion, migration, and inhibited BC cell proliferation. This evidence concerns the gene TP73 and breast carcinoma.