In the inflammatory process, GM‐CSF serves as a communication conduit between tissue‐invading lymphocytes and myeloid cells, and GM‐CSF‐activated phagocytes are well equipped to cause tissue damage.35 Chen et al36 reported that GM‐CSF‐treated cancer cells exhibited an enhanced ability of motility both in vitro and in vivo, and that chronic exposure of intestinal cancer cells to GM‐CSF led to the occurrence of intestinal epithelium to mesenchymal transition (EMT). The gene discussed is CSF2; the disease is intestinal cancer.