Furthermore, many hnRNPs, which are genetically associated with FTD/ALS, such as hnRNPA1, A2B1, and FUS (hnRNPP2) are reported to be involved in DNA damage and repair [4, 12, 24, 44, 48, 55], and hnRNPA3 is a homolog of hnRNPA1 and A2B1 [9, 59]. This evidence concerns the gene FUS and amyotrophic lateral sclerosis.