We have previously revealed that BMX kinase activity plays an important role in TNF‐induced EC angiogenesis 12 and VEGF‐induced lymphangiogenesis.16 With the development of specific BMX tyrosine kinase inhibitors, the inhibition of BMX may represent a feasible approach to synergistically attack tumour cells and tumour‐associated angiogenesis.17 The gene discussed is VEGFA; the disease is neoplasm.