Although further studies are deemed necessary to clarify how this interaction affects the distinct LPS-dependent signaling cascades, it is undoubted that the identification of an interplay between bindarit and FABP4 can open new perspectives for the exploitation of bindarit in the treatment of diseases where FABP4 plays a pivotal role, such as insulin resistance, type 2 diabetes, atherosclerosis and carcinogenesis55. This evidence concerns the gene FABP4 and atherosclerosis.