MYC and neoplasm: In the present study, we showed that knocking-down CCL14 could increase p-GSK3β (S9), β-catenin (S33/S37), and further promote the expression of c-myc and cyclin D1, which are the downstream target genes of Wnt/β-catenin and associated with tumor cell proliferation or apoptosis in various cancers26–28.