MYCN and medulloblastoma: Earlier reports emphasized the role of somatic MYC and MYCN gene amplification as a prevalent driver of medulloblastoma, but recent analyses of the whole genome sequencing data for medulloblastoma, in which all chromosomal rearrangements were considered, led to the identification of a novel region with highly disparate structural variant classes—i.e., focal deletion, tandem duplication, and inversion—in chromosome 9q34.13.