Although we did not address SeChry effects in CBS inhibition in our cell models, we can speculate that this effect is stronger in ovarian cancer cells, as CBS was reported to be involved in the promotion of ovarian tumor growth, cisplatin resistance, and cellular bioenergetics [27], as well as in the regulation of mitochondria morphogenesis, promoting tumor progression in ovarian cancer [26]. The gene discussed is CBS; the disease is ovarian carcinoma.