WEE1 and autoimmune thrombocytopenic purpura: It has been suggested that the pathogenesis of ITP was associated with gene expression, regulation of apoptosis, regulation of cell proliferation, nucleoplasm, transcription factor binding, histone deacetylase binding, protein kinase binding, and core promoter binding (Deng et al., 2017; Zuo et al., 2017), all of which were significantly enriched in the present study.