Furthermore, a novel subset of cultured neutrophils from patients with sepsis, CD16highCD24high, was shown to be significantly decreased because of the downregulation of the membrane CD24, and this effect was associated with the lack of neutrophil function in these patients, leading to an impairment in CD24‐mediated apoptosis of neutrophils and formation of a neutrophil extracellular trap 22. The gene discussed is CD24; the disease is Sepsis.