STBL was inversely correlated with inflammatory markers including C-reactive protein, neutrophil to lymphocyte ratio, and red cell distribution in patient with coronary atherosclerosis.[34] In addition, bilirubin partly inhibits the induction of complement through anti-apoptosis,[35] regulates the activity of various T lymphocytes,[36] and reduces the production of proinflammatory cytokines.[37]. Here, CRP is linked to coronary atherosclerosis.