Expression of the anti-apoptotic proteins Bcl-2 and Bcl-XL was upregulated by stress hormones and terbutaline, while the levels of cleaved caspase-3 and the pro-apoptotic protein Bax in GC cells were induced by non-selective ADRB blockers and specific ADRB2 blockers (Supplementary Fig. 5C). Here, BCL2 is linked to gastric cancer.