Moreover, high SNHG12 levels were also correlated with low levels of miR-133b (47) and miR-195 (48), which are known to be involved in regulating proliferation, migration, invasion, and the cell cycle of PCa by enhancing the p53 (47) and Wnt/β-catenin signaling pathways (48). The gene discussed is SNHG12; the disease is posterior cortical atrophy.