To better understand why the murine cancer cell lines expressed higher levels of Daxx at resting state than the human cancer cell lines, we examined the expression levels of HSP27 and its murine homolog, HSP25, in the human and murine cancer cell lines, respectively, because HSP27 has been previously shown to be a common binding partner of both p53 and Daxx in human cells27. The gene discussed is HSPB1; the disease is cancer.