Another study found that intravenous human MSCs improved myocardial infarction in mice without significant engraftment through the secretion of anti‐inflammatory protein TSG‐6.64 The mechanism of wound repair by MSCs has been suggested to stem from their paracrine signaling,65 which mobilizes the host cells to promote wound repair. The gene discussed is TNFAIP6; the disease is myocardial infarction.