This leads to stimulation of lipoprotein lipase (LPL), and increases plasma triglyceride clearance, conversion of the VLDL to LDL, and reducing the synthesis of LDL and reducing postprandial lipemia (Pizzini et al., 2017[88]; Chan et al., 2002[14], 2003[15]; Park and Harris, 2003[85]; Robinson and Stone, 2006[97]). The gene discussed is LPL; the disease is hyperlipidemia.