Overexpression of miR-96-5p was correlated with increased proliferation and migration by suppressing Caveolae1 (CAV1) and inhibiting AKT signaling pathway and its downstream proteins (Cyclin D1 and P70), thus implying that miR-96-5p could serve as a promising therapeutic target for ovarian cancer (Liu et al., 2019, p. 1). Here, AKT1 is linked to ovarian cancer.