We found that a 24h TGF-β1 stimulus generates gene expression changes related to regulatory pathways and transcriptional factors (MAPK, SREBP, and daunorubicin-associated pathways) while the longer-term stimulus provoked changes related to the fibrotic phenotype of fibroblasts, previously described in IPF, such as increased migration and secretion of matrix molecules and decreased proliferation [34]. This evidence concerns the gene TGFB1 and idiopathic pulmonary fibrosis.