To verify whether Mcl-1 deletion indeed resulted in an efficient neutropenia model in the context of atherosclerosis, Ldlr−/− recipient mice were transplanted with LysMCre Mcl-1fl/fl (hereafter Mcl-1−/−) or wild type (WT) bone marrow (Fig. 1E). This evidence concerns the gene LDLR and atherosclerosis.