However, plasma cystatin C, urine NGAL, and urine [TIMP-2]·[IGFBP7] levels at the time of admission were significantly increased in the AKI group compared to the non-AKI group (Cystatin C: 1.18 ± 1.6 vs. 0.99 ± 0.25 mg/dL, p = 0.01, NGAL 261.62 ± 507 vs. 56.85 ± 122.6 μg/gCr, p = 0.007, [TIMP-2]·[IGFBP7] 0.31 ± 1.09 vs. 0.1 ± 0.48, p = 0.019) (Table 1). This evidence concerns the gene LCN2 and acute kidney injury.