PRMT7 and cryptorchidism: The potential involvement of PRMT7 in neural function has been proposed by a recent study reporting a male with severe intellectual disability, facial dysmorphism, microcephaly, short stature, brachydactyly, cryptorchidism and seizures who was found to have a homozygous deletion of 15,309 base pairs encompassing the transcription start site of PRMT7, resulting in functionally a null allele17.