PREX1 and autism: Considering the expression of DIP2A in embryonic and early postnatal days, we speculate that DIP2A also affects other processes of neuronal development, such as the function of the autism-associated gene phosphatidylinositol 3,4,5-trisphosphate-dependent Rac exchanger 1 (P-Rex1) in synaptic transmission and neuronal migration [69,70].