Within the cohort of 173 receptor‐positive tumours, the presence of a p63/p40+/SMA− cell population did not correlate with HER2 status, TP53 mutation, tumour grade, lymph node status, proliferation using different Ki67 cut‐offs, or Nottingham prognostic index (NPI), but patients were more likely to be disease free at the time of last follow up (Table 1). This evidence concerns the gene TP53 and neoplasm.