Many EMT inducers act directly or indirectly by repressing E-cadherin, increasing vimentin and α–smooth muscle actin (αSMA) [3], a set of incidences that promote accumulation of myofibroblasts in injured tissues, which characterizes fibrotic diseases [5], including idiopathic pulmonary fibrosis (IPF). Here, VIM is linked to idiopathic pulmonary fibrosis.