A ubiquitin‐like domain and nucleolar localization signal are included in the N‐terminal and C‐terminal regions of MIDN, respectively, and MIDN is assumed to be involved in neurogenesis regulation.7 Since its discovery, it has been reported that the ubiquitin‐like domain interacts with glucokinase and that MIDN overexpression inhibits insulin secretion from MIN6 cells.8MIDN was also identified as a candidate gene implicated in female autism spectrum disorders.9 The gene discussed is MIDN; the disease is autism spectrum disorder.