STIP1 and infection: We hypothesized that if STI-1 compensates for the embryonic neocortical cells proliferation under FOXC1 inactivation, co-culture of STI-1-overexpressed mAPSCs produced by LV-STI-1 infection (LV-STI-1-mAPSCs) with FOXC1-/-embryonic neocortical neurospheres would enhance the proliferative potential of neurospheres.