As SK channels appears to play a role in some pathological conditions in the ventricles (Gui 2013; Bonilla 2014; Hundahl 2017; Chen 2018; Hamilton 2019), it could be speculated that the SK channels also have a contributory role in the pathogenesis of CaM‐associated LQTS. The gene discussed is CALM2; the disease is familial long QT syndrome.