COL1A2 and myocardial infarction: Loss of fibroblast MCU (Mcufl/fl × Col1a2-CreERT) significantly increased LV dilation, evident by increased LV end-diastolic diameter (LVEDD) and end-systolic diameter (LVESD), as well as reduced fractional shortening (FS) 2–4 weeks post-MI, as compared to Col1a2-CreERT controls (Fig. 6d–f).