To further assess the effect of myeloid deficiency of PPAR-γ on recovery following influenza infection, we examined lesions and collagen deposition in the lung tissue at 60 days p.i. H&E staining of lung tissue sections revealed sustained inflammation and damage from mice with PPAR-γ deficient macrophages compared to those of WT lungs that were largely resolved in these regards at 60 days p.i. (Fig 4A). The gene discussed is PPARG; the disease is influenza.