Akt1 is related to proliferation and antiapoptotic behavior.4 Akt2 deletion leads to hyperglycemia, a type‐2 diabetic phenotype, and the impairment of glucose uptake.5 Absence of Akt3 results in neuronal malfunction and altered fatty acid metabolism.6 Recent knock‐down studies underlined the opposing roles of Akt1 and Akt2 in different cancer types. Here, AKT1 is linked to Hyperglycemia.