Other works carried out in triple negative breast cancer cells (TNBCs) have shown the acquisition of resistance to be associated with the hyperphosphorylation state of BRD4 as a consequence of a reduction in the expression of the BRD4 phosphatase, PP2A, and a concomitant enrichment in the levels of the transcriptional activator associated with BRD4, MED1 [103]. Here, BRD4 is linked to triple-negative breast carcinoma.