Qualitative RT-PCR analysis revealed that G-Hes treatment downregulated mRNA expressions of Saa3, C/EBPβ, ColI, αSMA, and TNF-α (Fig. 4g–k), indicating a possible role of G-Hes in suppressing inflammation and fibrosis in adenine-induced kidney disease. The gene discussed is CEBPB; the disease is kidney disorder.